ECG Case Series
ECG case series for paramedics: September 2024
Charles Bloe, Coronary Care Unit, NHS Highland, Inverness, UK. Email: charles.bloe@nhs.scot
This 94-year-old man lives in a care home. He has had multiple hospital admissions over the last 2 years for general frailty and urinary tract problems. He is known to have prostate cancer that is being medically managed and has a permanent urinary catheter in situ. He had recently been in hospital for treatment of a urinary tract infection (UTI).
This morning, he complained of being short of breath at rest. He is normally mobile using a walking frame but today, he is unable to walk. His oxygen saturation was 89% on air. His pulse was also noted to be irregular. The last time this was checked, he had a normal, regular pulse. The staff in the care home decided to call for an ambulance.
His vital signs were as follows: l Blood pressure: 98/58 mmHg l Pulse: 86 beats per minute, with a very irregular pulse noted l Respiratory rate: 20 breaths per minute l He was apyrexial l SpO2 on air: 89%.
Figure 1. ECG of a 94-year-old man who found himself short of breath at rest.
The 12-lead electrocardiogram (ECG) in Figure 1 was recorded:
Interpretation of the ECG rhythm l The heart rhythm is regular l The heart rate is approximately 75 beats per minute l The rhythm is very irregular l The heart rate is approximately 80 beats per minute l There are no identifiable P waves l V1 in particular shows sawtooth flutter waves
(hint: if you are struggling to see P waves on an ECG, have a look at V1 and V2 where atrial activity is often easier to see) l The QRS complexes are >0.10 seconds in duration, i.e. wide l There is a left bundle branch block (LBBB)
configuration (RSR complexes in lead I, aVL, V5 and V6) l The cardiac axis is normal (positive QRS
complexes in leads I and aVF). This ECG shows the patient to be in atrial flutter with a variable ventricular response and left bundle branch block configuration. His high sensitivity troponin was moderately elevated.
A final diagnosis of new onset atrial flutter and LBBB with subsequent heart failure was made.
LBBB causes an abnormal depolarisation. This also results in abnormal repolarisation, which causes a secondary ST/T wave abnormality. A primary ST/T wave abnormality would be caused by myocardial infarction. LBBB results in secondary ST/T wave abnormalities that can mimic ST segment elevation myocardial infarction (STEMI). In this case, there is appropriate ST segment and T wave discordance, i.e. in those leads with a positive QRS complex, the ST segment and T wave are deflected in the opposite direction and vice versa. This is an appropriate finding in LBBB and is a secondary ST/T wave abnormality, i.e. not ischaemia/infarction. JPP
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358 https://doi.org/10.12968/jpar.2024.0071
Vol 16 No 9 • Journal of Paramedic Practice